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While EECp failed to downsize bladder edema with the comparable efficiency of mesna, it enhanced significantly the histopathological scores and diminished myeloperoxidase activity in the lung, as well as, the levels of malondialdehyde in the bladder, lung, and spleen tissue

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Ndufs2 KO adipocytes showed reduced expression of the pathways involved in fatty acid metabolism, insulin signalling, peroxisome proliferator-activated receptor (PPAR) signalling and lipolysis, along with activation of oxidative stress, inflammatory and glycolytic programmes