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Table 2 provides a comprehensive catalog of the characterized PSMD14 substrates, their deubiquitination types, downstream biological effects, and associated drug resistance profiles across different malignancies, offering a molecular rationale for the inhibitor strategies discussed above

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Jun-terminal kinases (JNKs) are a sub-class of MAPK kinases, initially identified as stress-activated protein kinases in mouse liver treated with cycloheximide to induce inflammation and apoptosis [58]